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Table 3. Independent predictors of complicated course in patients with CAP 19 Predictor Age 65 Years Comorbid illness Temperature 38C Immunosuppressive therapy * High-risk etiology * Recent systemic steroid use or cancer chemotherapy Odds ratio 2.7 3.2 4.1 Confidence.Interval 1.4 - 4.1 1.4 - 7.5 1.8 - 9.2 1.1 - 132.9 2.7 - 200.7.

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Therapy of HCV. Treatment of HCV that results in viral clearance normalization of alanine aminotransferase ; has been shown to reduce the rate of progression to cirrhosis and hepatocellular carcinoma. Several newer therapeutic strategies are undergoing clinical investigation 364 ; . These consist of modifications of IFN to improve efficacy and reduce side effects, including conjugation with albumin, liposome encapsulation, and polyamino acidbased oral delivery systems. Other potential agents include broad-spectrum antiviral agents, inhibitors of HCV NS3 serine protease, RNA-dependent DNA polymerase inhibitors, ribozymes that cleave specific RNA sequences, antisense oligonucleotides that recognize the noncoding region of HCV RNA, monoclonal antibodies against HCV envelope protein, and agents, such as thymosin a-1 and the natural killer cell activator histamine dihydrochloride, to enhance immune activity 365 ; . The acyclic retinoid, polyprenoic acid 3, 7, 11, acid ; , inhibits chemically induced hepatic carcinogenesis in rats and spontaneous hepatocellular carcinoma in mice and suppresses human hepatoma cell growth and a-fetoprotein production in vitro. The exact mechanism of action of polyprenoic acid is uncertain; however, preclinical studies suggest that this agent can induce both differentiation and apoptosis of hepatocellular carcinoma cells. A 12-month course of oral polyprenoic acid significantly reduced both recurrent and second primary hepatomas at 38 months; however, follow-up was insufficient to establish an effect on survival 366 ; . A phase II III study of polyprenoic acid as adjuvant therapy following surgery or ablation of hepatocellular carcinoma in HCV patients is currently under way. Several studies investigated the expression of COX-2 in human hepatocellular carcinoma to evaluate the possible use of COX-2 inhibitors in the prevention and treatment of this malignancy 367 ; . COX-2 was overexpressed in 75% to 100% of hepatocellular carcinoma specimens and in 47% to 70% of the adjacent noncancerous tissue; well-differentiated hepatocellular carcinoma expresses COX-2 more frequently and strongly than less differentiated tumors. A significant increase in COX-2 levels occurs in liver tissue in parallel with disease progression from chronic hepatitis to cirrhosis 368 ; . Hepatitis B protein HBx, often the only HBV protein detected in HBV-associated hepatocellular carcinoma, induced COX-2 369 ; . Preclinical evidence supports the potential use of COX-2 inhibitors to reduce the risk of hepatocellular carcinoma. Specifically, the COX-2 inhibitor JTE-522 was highly effective in reducing the development of both cirrhosis and hepatocellular carcinoma in a rat model 370 ; . These agents have not yet been thoroughly evaluated in human studies. Hepatocellular carcinoma occurs largely in individuals with known risk factors, and significant reduction in hepatocellular carcinoma incidence has been shown through prevention of HBV transmission. In this respect, hepatocellular carcinoma is unique in contrast to most other major cancers, for which risk factors can only be identified at the population level. Thus, hepatocellular carcinoma represents a prime example of an incurable cancer that can indeed be prevented. To advance prevention of hepatocellular carcinoma, clinical trials are needed in the following areas: a ; more effective HBV therapies; b ; new agents to increase the HCV sustained viral response, and identification of factors associated with lack of, for instance, amoxicillin.

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Administration of clarithromycin with ergotamine or dihydroergotamine is contraindicated See CONTRAINDICATIONS ; . Triazolobenziodidiazepines such as triazolam and alprazolam ; and related benzodiazepines such as midazolam ; : Erythromycin has been reported to decrease the clearance of triazolam and midazolam, and thus, may increase the pharmacologic effect of these benzodiazepines. There have been postmarketing reports of drug interactions and CNS effects e.g., somnolence and confusion ; with the concomitant use of clarithromycin and triazolam. HMG-CoA Reductase Inhibitors: As with other macrolides, clarithromycin has been reported to increase concentrations of HMG-CoA reductase inhibitors e.g., lovastatin and simvastatin ; . Rare reports of rhabdomyolysis have been reported in patients taking these drugs concomitantly. Sildenafil Viagra ; : Erythromycin has been reported to increase the systemic exposure AUC ; of sildenafil. A similar interaction may occur with clarithromycin; reduction of sildenafil dosage should be considered. See Viagra package insert. ; There have been spontaneous or published reports of CYP3A based interactions of erythromycin and or clarithromycin with cyclosporine, carbamazepine, tacrolimus, alfentanil, disopyramide, rifabutin, quinidine, methylprednisolone, cilostazol, and bromocriptine. Concomitant administration of clarithromycin with cisapride, pimozide, astemizole, or terfenadine is contraindicated see CONTRAINDICATIONS ; . In addition, there have been reports of interactions of erythromycin or clarithromycin with drugs not thought to be metabolized by CYP3A, including hexobarbital, phenytoin, and valproate. For information on interactions between clarithromycin in combination with other drugs which may be administered to HIV-infected patients, see the BIAXIN package insert, Drug Interactions, under the PRECAUTIONS section. Drug Laboratory Test Interactions High urine concentrations of ampicillin may result in false-positive reactions when testing for the presence of glucose in urine using Clinitest, Benedict's Solution or Fehling's Solution. Since this effect may also occur with amoxicillin, it is recommended that glucose tests based on enzymatic glucose oxidase reactions such as Clinistix ; be used. Following administration of ampicillin to pregnant women, a transient decrease in plasma concentration of total conjugated estriol, estriol-glucuronide, conjugated estrone, and estradiol has been noted. This effect may also occur with amoxicillin. Carcinogenesis, Mutagenesis, Impairment of Fertility PREVACID: In two 24-month carcinogenicity studies, Sprague-Dawley rats were treated orally with doses of 5 to 150 mg kg day, about 1 to 40 times the exposure on a body surface mg m2 ; basis, of a 50-kg person of average height 1.46 m2 body surface area ; given the recommended human dose of 30 mg day 22.2 mg m2 ; . Lansoprazole produced dose-related gastric enterochromaffin-like ECL ; cell hyperplasia and ECL cell carcinoids in both male and female rats. It also increased the incidence of intestinal metaplasia of the gastric epithelium in both sexes. In male rats, lansoprazole produced a dose-related increase of testicular interstitial cell adenomas. The incidence of these adenomas in rats receiving doses of 15 to 150 mg kg day 4 to 40 times the recommended human dose based on body surface area ; exceeded the low background incidence range 1.4 to 10% ; for this strain of rat. Testicular interstitial cell adenoma also occurred in 1. 166 REFERENCES Billard, R., Alagarswami, K., Peter, R. E. and Breton, B., 1983. Potentialisation par le pimozide des effects du LH-RH-A sur la s&r&ion gonadotrope hypophysaire, l'ovulation et la spermiation chez la carpe commune Cyprinus carpio ; . C.R. Acad. Sci Paris ; Ser. C., 296: 181-184. Breton, B., Jalabert, B., Bieniarz, K., Sokolowska, M. and Epler, P., 1983. Effect of synthetic LHRH and analog on plasma gonadotropin levels and maturational response to 17a-hydroxy20 ?-dihydroprogesterone. Aquaculture, 32: 105-114. Chang, J. P. and Peter, R. E., 1983. Effects of pimozide and des-Gly"' [D-Ala'] luteinizing hormone-releasing hormone ethylamide on serum gonadotropin concentrations, germinal vesicle migration and ovulation in female goldfish, Carassius auratus. Gen. Comp. Endocrinol., 52: 30-37. Chang, J. P., Peter, R. E., Nahorniak, C. S. and Sokolowska, M., 1984. Effects of catecholaminergic agonists and antagonists on serum gonadotropin concentrations and ovulation in goldfish: evidence for specificity of dopamine inhibition of gonadotropin secretion. Gen. Comp. Endocrinol., 55: 351-360. Colombo, L., Colombo-Belvt%re, P. and Arcaresse, G., 1978. Emergence of ovarian 1 l-deoxycorticosteroid biosynthesis at ovulation time in the sea bass, Dicentrarchus labrax L. Ann. Biol. Anim. Biochim. Biophys., 18: 937-941. De Leeuw, R., Resink, J. W., Rooyakkers, E. J. M. and Goos, H. J. Th., 1985a. Pumozide modulates the luteinizing hormone-releasing hormone effect on gonadotropin release in the African catfish, Clarias lazera. Gen. Comp. Endocrinol., 58: 120-127. De Leeuw, R., Goos, H. J. Th., Richter, C. J. J. and Eding, E. H., 1985b. Pimozide-LHRHainduced breeding of the African catfish, Clariasgariepinus Burchell ; . Aquaculture, 44: 295-302. De Leeuw, R., Goos, H. J. Th. and Van Oordt, P. G. W. J., 1986. The dopaminergic inhibition of the gonadotropin-releasing hormone-induced gonadotropin release: an in vitro study with fragments and cell suspensions from pituitaries of the African catfish, Clarias gariepinus Burchell ; . Gen. Comp. Endocrinol., 63: 171-177. De Leeuw, R., Goos, H. J. Th. and Van Oordt, P. G. W. J., 1987. The regulation of gonadotropin release by neurohormones and gonadal steroids in the African catfish, Clark gariepinw Burchell ; . Aquaculture, 63: 43-58. Donaldson, E. M. and Hunter, G. A., 1983. Induced final maturation, ovulation and spermiation in cultured fish. In: W. S. Hoar, D. J. Randall andE. M. Donaldson Editors ; , Fish Physiology. Vol. IXB, Reproduction. Academic Press, New York, NY and London, pp. 351-403. Duffey, R. J. and Goetz, F. W., 1980. The in vitro effect of 17a-hydroxy-20 I-dihydroprogesterone on germinal vesicle breakdown in brook trout Saluelinus fontinalis ; oocytes. Gen. Comp. Endocrinol., 41: 563-565. Eding, E. H., Janseen, J. A. L., Kleine Staarman, G. H. J. and Richter, C. J. J., 1982. Effects of human chorionic gonadotropin HCG ; on maturation and ovulation of oocytes in the ovary of the African catfish, Clark &era, In: C. J. J. Richter and H. J. Th. Goos Editors ; , Proc. Int. Symp. Reproductive Physiology of Fish, Wageningen, The Netherlands, 2-6 August 1982. PUDOC, Wageningen, p. 195. Epler, P., 1981a. Effect of steroid and gonadotropic hormones on the maturation of carp ovaries. II. Effect of fish and mammalian gonadotropins on the maturation of carp oocytes in vitro. Pol. Arch. Hydrobiol., 28: 103-110. Epler, P., 1981b. Effect of steroid and gonadotropic hormones on the maturation of carp ovaries. IV. A combined action of steroid hormones and HCG or fish hypophysial homogenate on carp oocyte maturation in vitro. Pol. Arch. Hydrobiol., 28: 111-117. Fostier, A., Jalabert, B. and Terqui, M., 1973. Action predominante d'un derive hydroxyle de la progesterone sur la maturation in vitro des oocytes de la Truite arc-en-ciel, Salmo gairdneri. C. R. Hebd. Seances Acad. Sci., 277: 421-424 and ondansetron.

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Clozapine and pimozide are the only antipsychotics that should be avoided when co-administered with the protease inhibitor ritonavir because of cytochrome p450 interactions.
168 spawning behaviour and social stimuli in a wild population of rainbow trout Salmo gairdneri ; . II. Females. Gen. Comp. Endocrinol., 62: 157-167. Lin, H. -R., Peng, C., Lu, L. Z., Zhou, X. -J., Van Der Kraak, G. and Peter, R. E., 1985. Induction of ovulation in the loach Paramisgurnus dabryanus ; using pimozide and [ D-Ala', Pro'-Nethylamide] -LHRH. Aquaculture, 46: 333-340. Montalembert, G., Jalabert, B. and Bry, C., 1978. Precocious induction of maturation and ovulation in northern pike Esox Lucius ; . Ann. Biol. Anim. Biochim. Biophys., 18: 969-975. Nagahama, Y., Hirose, K., Young, G., Aduchi, S., Suzuki, K. and Tamaoki, B., 1983. Relative in vitro effectiveness of 17a, 20 -dihydroxy-4-pregnene-3-one and other pregnene derivatives on germinal vesicle breakdown in oocytes of four species of teleosts, ayu Plecoglossus altiuelis ; , amago salmon Oncorhynchus rhodurus ; , rainbow trout Salmo gairdneri ; and goldfish Carassius auratus ; . Gen. Comp. Endocrinol., 51: 15-23. Peter, R. E. and Crim, L. W., 1978. Hypothalamic lesions of goldfish: effect on gonadal recrudescence and gonadotropin secretion. Ann. Biol. Anim. Biochim, Biophys., 18: 819-823. Peter, R. E. and Paulencu, C. R., 1980. Involvement of the preoptic region in gonadotropin release inhibition in goldfish, Carassius auratus. Neuroendocrinology, 31: 133-141. Peter, R. E., Crim, L. W., Goos, H. J. Th. and Crim, J. W., 1978. Lesioning studies on the gravid female goldfish: neuroendocrine regulation of ovulation. Gen. Comp. Endocrinol., 35: 391-401. Richter, C. J. J. and Van Den Hurk, R., 1982. Effects of ll-desoxycorticosterone-acetate and carp pituitary suspension on follicle maturation in the ovaries of the African catfish, Clarias lazera C. &V. ; . Aquaculture, 29: 53-66. Richter, C. J. J., Eding, E. H. and Bloem, A. J., 1985.17c + Hydroxyprogesterone-induced breeding of the African catfish, Clark gariepinus Burchell ; , without priming with gonadotropin. Aquaculture, 44: 285-293. Schoonen, W. G. E. J., Lambert, J. G. D., Resink, J. W., Viveen, W. J. A. R. and Van Oordt, P. G. W. J., 1987. A quantitative study of steroid bioconversion in the testes of the African catfish, Clarias gariepinus Burchell ; under natural spawning and non-spawning conditions, and under fish farming conditions. J. Endocrinol., 112: 323-332. Scott, A. P., Sheldrick, E. L. and Flint, A. P. F., 1982a. Measurement of 17a, 20P-dihydroxy-4pregnene-3-one in plasma of trout Salmo gairdneri Richardson ; : seasonal changes and response to salmon pituitary extract. Gen. Comp. Endocrinol., 46: 444-451. Scott, A. P., Sumpter, J. P. and Hardiman, P. A., 1982b. Hormone changes during ovulation in the rainbow trout Salmo gairdneri ; . Gen. Comp. Endocrinol., 49: 128-134. Scott, A. P., MacKenzie, D. S. and Stacey, N. E., 1984. Endocrine changes during natural spawning in the white sucker, Catostomus commersonii. II. Steroid hormones. Gen. Comp. Endocrinol., 56: 349-359. Sokolowska, M., Peter, R. E., Nahorniak, C. S., Pan, C. H., Chang, J. P., Crim, L. W. and Weil, C., 1984. Induction of ovulation in goldfish, Carassius auratus, by pimoziee and analogues of LHRH. Aquaculture, 36: 71-83. Stacey, N. E. and Pandey, A., 1975. Effect of indomethacin and prostaglandin on ovulation of goldfish. Prostaglandins, 9: 597-607. Sundararaj, B. I. and Goswami, S. V., 1977. Hormonal regulation of in vivo and in vitro oocyte maturation in the catfish, Heteropneustus fossilis Bloch ; . Gen. Comp. Endocrinol., 32: 17-28. Van Der Kraak, G., Dye, H. M., Donaldson, E. M. and Hunter, G. A., 1985. Plasma gonadotropin, 17-p-estradiol, and 17o, 20 ?-dihydroxy-4-pregnene-3-one levels during luteinizing hormonereleasing hormone analogue and gonadotropin induced ovulation in coho salmon Oncorhynthus kisutch ; . Can. J. Zool., 63: 824-833. Van Ree, G. E., Lok, D. and Bosman, G., 1977. In vitro induction of nuclear breakdown in oocytes of the zebrafish, Brachydanio rerio Ham-Buch ; . Effects of the composition of the medium and of protein and steroid hormones. Proc. K. Ned. Akad. Wet., Ser. C, 80: 353-371. Young, G., Kagawa, H. and Nagahama, Y., 1982. Oocyte maturation in the amago salmon Oncorhynchus rho&us ; : in vitro effects of salmon gonadotropin, steroids and cyanoketone an inhibitor of 3 3-hydroxy-A5-steroid dehydrogenase. J. Exp. Zool., 224: 265-275 and zofran.

4 0 comment didn't notice any benefit i really didn't notice much if any ; effect on my skin while using this medication, for instance, haldol. Composition: Tris . 53.91 g L 0.45 M ; EDTA-Na 2 H2O . 3.72 g L 0.10 M ; Boric acid . 27.52 g L 0.45 M ; Specification: DNases RNases . not detectable pH 20C, H2O ; . 8.3 0.2 and oxcarbazepine. Pharmacy providers automatically receive a Durable Medical Equipment DME ; location code when they first enroll. To be reimbursed for DME and medical supplies, the pharmacy provider must request activation of the location code by sending a letter to the Medicaid fiscal agent to request activation of the DME locator code. The DME locator code attached to the pharmacy must be at the same location as the pharmacy. The letter must contain an original signature. Faxed letters will not be accepted. Mail the letter to: ACS Provider Enrollment P.O. 7070 Tallahassee, Florida 32314-7070 All DME billing must be on the CMS-1500 claim form using the pharmacy's provider number with the unique DME locator code.
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Usa address of corresponding author american journal of nephrology 2004; 1-648 doi: 1 1159 000082946 ; key words angiotensin ii diltiazem pimozife voltage-dependent calcium channels calcium channel blockers t-type ca 2 + channels l-type ca 2 + channels renal microcirculation kidney abstract background aims: previous studies have shown that l-type ca 2 + channel lcc ; blockers prevent the afferent arteriolar aa ; vasoconstriction elicited by angiotensin ii ang ii ; , but do not influence its vasoconstrictor effect on efferent arterioles ea and trileptal. CYP3A4 substrates Roxithromycin is a weak inhibitor of CYP3A4. Concomitant intake of roxithromycin and midazolam increased the midazolam a sensitive CYP3A4 substrate ; AUC by 47%. Although roxithromycin is a weak CYP3A inhibitor, a possible clinical relevant inhibition of this enzyme can not be excluded in some individuals, therefore co-administration with other medicinal products with narrow therapeutic window metabolised by CYP3A e.g. ciclosporin, ergot alkaloid derivates, terfenadine, cisapride, pimozide, astemizole ; is not recommended see also section 4.3 ; Ergot alkaloid derivatives such as ergotamine and dihydroergotamine ; Concomitant medication of roxithromycin and alkaloid derivatives could lead to severe vasoconstriction "ergotism" ; with possibly necrosis of the extremities. The combination is contraindicated see section 4.3. 1. Drucker DJ 1998 Glucagon-like peptides. Diabetes 47: 159 169 Drucker DJ, Philippe J, Mojsov S, Chick WL, Habener JF 1987 Glucagon-like peptide I stimulates insulin gene expression and increases cyclic AMP levels in a rat islet cell line. Proc Natl Acad Sci USA 84: 3434 3438 Mojsov S, Weir GC, Habener JF 1987 Insulinotropin: glucagon-like peptide I 737 ; co-encoded in the glucagon gene is a potent stimulator of insulin release in the perfused rat pancreas. J Clin Invest 79: 616 619 Kreymann B, Williams G, Ghatei MA, Bloom SR 1987 Glucagon-like peptide-1 736: a physiological incretin in man. Lancet 2: 1300 1304 Ritzel R, Orskov C, Holst JJ, Nauck MA 1995 Pharmacokinetic, insulinotropic, and glucagonostatic properties of GLP-1 [736 amide] after subcutaneous injection in healthy volunteers. Dose-response-relationships. Diabetologia 38: 720 725 Schirra J, Katschinski M, Weidmann C, Schager T, Wank U, Arnold R, Goke B 1996 Gastric emptying and release of incretin hormones after glucose ingestion in humans. J Clin Invest 97: 92103 7. Turton MD, O'Shea D, Gunn I, Beak SA, Edwards CM, Meeran K, Choi SJ, Taylor GM, Heath MM, Lambert PD, Wilding JP, Smith DM, Ghatei MA, Herbert J, Bloom SR 1996 A role for glucagon-like peptide-1 in the central regulation of feeding. Nature 379: 69 72 and oxytetracycline and pimozide, for example, pregnancy.
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Concern that these drugs, in particular, may provoke arrythmias and sudden death. Regulatory action has previously been taken for pimoaide and sertindole, and we recently demonstrated that prolongation of the QT interval is more common with thioridazine and droperidol than with other antipsychotic drugs Reilly et al, 2000 ; . However, the al, systematic evidence in support of a direct link between antipsychotic drug therapy and arrythmia or sudden cardiac death is sparse and orinase.
External customers. BASF sells its chemicals to a multitude of industries, particularly the chemical, construction, automotive and electronics industries. Plastics & Fibers BASF is one of the world's largest plastics and ber products manufacturers. Its products include styrenic plastics, engineering and high-performance plastics, thermoplastics, foams, nylon bers and polyurethane plastics. Colorants & Finishing Products The Colorants & Finishing Products segment manufactures a number of BASF's high-value chemical products. Among the segment's products are colorants, pigments, automotive and industrial coatings as well as adhesives. BASF also manufactures superabsorbents, which are used to manufacture sanitary care products. Health & Nutrition: Pharmaceuticals, Fine Chemicals and Crop Protection BASF is active in the areas of pharmaceuticals, ne chemicals and crop protection. In pharmaceuticals, BASF is focused on therapeutic areas with high medical need and large patient populations. BASF is also a leading supplier of ne chemicals, including vitamins and other nutrients for human and animal nutrition. In addition, BASF produces a variety of crop protection products, including fungicides and herbicides. Oil & Gas The Oil & Gas segment operates through BASF's subsidiary Wintershall AG. The main activities of the Oil & Gas segment are the exploration and production of crude oil and natural gas and, together with Wintershall AG's partner Gazprom of Russia, the marketing, distribution and trading of natural gas in Central and Eastern Europe. Group Strategy Three key elements of BASF's strategy are: , generating long-term growth and protability from its chemical activities; , positioning itself for continuing growth in health and nutrition; and , strengthening its presence in European and other oil and gas markets. As an industry leader, BASF has set the following strategic goals: , Achieve signicant cost and operational production eciencies through its Verbund approach to integration BASF believes it is one of the world's most ecient chemical producers due to the highly integrated nature of its manufacturing sites. BASF's integrated approach, known in German as ""Verbund, '' generates signicant eciencies throughout BASF's production activities, which BASF believes historically has allowed the company to achieve higher operating returns than its less-integrated competitors. Verbund is powerfully represented at BASF's chemical manufacturing sites, which are among the largest in the world. At its headquarters site in Ludwigshafen, which serves as a model for Verbund, about 350 plants and processes are integrated vertically and horizontally in dozens of value-adding chains. This approach allows BASF to manufacture basic chemicals predominately for sale to other operations within the company. Although the transfer prices that these operations pay for internal purchases are typically based on market prices, Verbund oers internal buyers signicant savings in logistics and in energy, transportation, purchasing and infrastructure costs. BASF operations transform products that they buy internally 2.

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Many, if not most, biologically based medical conditions are diagnosed by the observation and experience of a trained clinician or physician. Antagonists that had no significant effect upon the action of 5HT and DA Antagonist Ergonovine NAN-190 Ketanserin Ritanserin MDL 72222 Tropisetron Bulbocapnine Butaclamol Ergonovine Apomorphine Chlorpromazine Fluphenazine Pimozids Spiperone Sulpiride Type * DA 5HT 5HT1A 5HT2 DA DA DA 5HT DA2 DA2 DA2 DA2 DA2 DA2 3 Agonist 5HT DA DA DA Mean contraction ratio 1.42 1.66 1.68 ; 1.52 6 ; 1.15 6 ; 1.67 7 ; 1.73 6 ; 0.680 6 ; 6.29 6 ; 1.60 17 ; 1.24 4 ; 4.93 7 ; 1.06 5 ; 1.61 6 ; 1.67 6 ; 0.812 16 ; 2.23 8 ; SD n ; 0.5 0.49 0.5.
Temporary Assistance to Needy Families Adults Only ; General Assistance PLM Adults PLM, TANF, and CHIP Children 1 PLM, TANF, and CHIP Children 1 - 5 PLM, TANF, and CHIP Children 6 - 18 OHP Families OHP Adults & Couples Aid to the Blind Aid to the Disabled with Medicare Aid to the Blind Aid to the Disabled without Medicare Old Age Assistance with Medicare Old Age Assistance without Medicare SCF Children CAWEM Citizen-Alien Waived Emergency Medical ; FCHP PER CAPITA RATE $27.47 $32.68 $13.92 $0.09 $11.97 $17.83 $30.46 $36.22 $23.42 $20.78 $15.01 $25.07 $17.16 $0.00 FCHP FFS PER CAPITA RATE $0.00 $0.00 $0.00 $0.00 $0.00 $0.00 $0.00 $0.00 $0.00 $0.00 $0.00 $0.00 $0.00 $0.00 FFS PCCM PER CAPITA RATE $2.06 $2.03 $0.57 $0.00 $0.75 $1.16 $1.62 $1.74 $1.40 $1.79 $0.81 $0.28 $1.75 $0.00.

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